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REVIEW
New targets for the therapy of Pulmonary
Arterial Hypertension (PAH)
Nebulized anti-miRs against miR-138 and miR-25 completely
regress monocrotaline-induced PAH
| 1 | Academic Department of Pneumonology,
Hospital for Diseases of the Chest, “Sotiria”,
Medical School, University of Athens,
Messogion Avenue 152, Athens, Greece |
| 2 | Division of Immunology, Biomedical Sciences
Research Center “Alexander Fleming”, Vari,
Athens, Greece |
CORRESPONDING AUTHOR
Argyris Tzouvelekis
Biomedical Sciences Research Center “Alexander Fleming”, Division of Immunology, Vari 16672, Athens, Greece
Biomedical Sciences Research Center “Alexander Fleming”, Division of Immunology, Vari 16672, Athens, Greece
Publication date: 2021-06-16
Pneumon 2016;29(3):236–237
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Benza RL, Miller DP, Barst RJ, Badesch DB, Frost AE, McGoon MD. An evaluation of long-term survival from time of diagnosis in pulmonary arterial hypertension from the REVEAL Registry. Chest 2012;142:448-56.
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Hong Z, Chen KH, Dasgupta A, et al. miR-138 and miR-25 Downregulate MCU, Causing Pulmonary Arterial Hypertension’s Cancer Phenotype. American Journal of Respiratory and Critical Care Medicine 2016.
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Santulli G, Xie W. Mitochondrial calcium overload is a key determinant in heart failure. 2015;112:11389-94.
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Yan H, Zhang D, Hao S, Li K, Hang CH. Role of Mitochondrial Calcium Uniporter in Early Brain Injury After Experimental Subarachnoid Hemorrhage. Molecular Neurobiology 2015;52:1637-47.
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